Acute kidney injury (AKI) affects up to 50% of critically ill patients and is independently associated with both short- and long-term morbidity and mortality [1–5]. The recent AKI-EPI study demonstrates that the most frequent causes of AKI in the critically ill are sepsis and hypovol-aemia followed by nephrotoxic agents [6]. However, the cause of AKI is often multifactorial with pre-existing co-morbidities further increasing the risk [3, 7–9].


Jaime Pabón

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